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Height, Gender, and Longevity: Can Tall Men Close the Gap with Shorter People and Women?

Hey there, longevity enthusiasts! If you’ve ever wondered why your grandma outlives your grandpa or why those pint-sized folks seem to stick around forever, you’re not alone. In a world obsessed with living longer, factors like gender and height play surprisingly big roles in our lifespans. As a tall guy myself (or at least, imagining I am for this post), I’ve dived into the data to compare tall men, short men, and women when it comes to longevity. We’ll break down the science, the disadvantages, and—most importantly—actionable ways for tall men to shrink that gap. Spoiler: It’s not all doom and gloom; with smart habits, you can stack the odds in your favor.

Based on the latest 2025 stats, women in the US outlive men by about 5 years on average—81 years for women versus 76 for men. 1 Globally, the gap is similar, around 5.2 years across OECD countries. 4 But throw height into the mix, and things get even more interesting. Studies show shorter people often have a longevity edge over their taller counterparts. 10 12 Let’s unpack this step by step.

The Gender Gap: Why Women Often Win the Longevity Race

Women have a built-in advantage when it comes to living longer, and it’s not just about avoiding risky jobs or bad habits (though those play a part). Biologically, women benefit from higher estrogen levels, which protect against heart disease by managing cholesterol and fat storage. 1 They also have a stronger immune system, thanks to two X chromosomes providing genetic backups against mutations. 1 Men, on the other hand, deal with higher testosterone, which can amp up risks for cardiovascular issues and even make diseases like COVID-19 more deadly.

Behaviorally, men historically delay doctor visits or engage in more high-risk activities, but even when we control for that, the gap persists. 3 In 2025, this translates to women enjoying about 2.7 more cancer-free years after age 50 compared to men. 2 But here’s a ray of hope: In “Blue Zones”—regions like Sardinia where people routinely hit 100—the gender gap can narrow or even disappear when men adopt ultra-healthy lifestyles. 33 30 More on that later.

The Height Hurdle: Tall vs. Short in the Longevity Game

Now, let’s talk stature. Multiple studies reveal a negative correlation between height and lifespan: The taller you are, the shorter your expected life, on average. 10 14 Why? Taller bodies have more cells, which means more opportunities for mutations leading to cancer or other diseases. 12 Plus, bigger frames demand more energy, putting extra strain on the heart, lungs, and joints over time. 15

• Tall Men (e.g., 6 feet or taller): Often face a 2-7 year disadvantage compared to shorter men. One study found men over 6 feet live shorter than those under 5’9”, with differences up to 5 years. 28 Risks include higher cancer rates and cardiovascular stress.
• Short Men (e.g., 5’6” or under): They tend to outlive taller guys by several years, thanks to lower disease risks and efficient body mechanics. 11 17 Shorter stature correlates with better resistance to age-related issues.
• Women (e.g., average 5’4” or shorter like 5’3”): Already ahead by gender, shorter women amplify this with height benefits. They often live 1-2 years longer than taller women, combining hormonal protections with reduced cellular risks. 12

In rural Spain cohorts, taller heights (over 170 cm) linked to longer lives in some historical contexts, but modern data flips this—shorter is better in today’s world. 13 18 For a 6-foot man vs. a 5’3” woman, the combined gap could be 10-11 years without interventions.

The Double Whammy for Tall Men: Stacking Gender and Height Against You

Imagine a tall man (6 feet, lean and healthy) versus a short man (5’6”) and a short woman (5’3”). The tall guy starts with a 5-year gender hit compared to the woman, plus 3-5 years from height alone versus the short man. 11 14 Total disadvantage: Up to 8-10 years shorter lifespan on average. Short men might close some of the gender gap but still lag behind women by 3-4 years. Women, especially shorter ones, sit at the top with the longest projections.

But these are averages from population studies—genetics, environment, and luck vary wildly. 10 The good news? Lifestyle tweaks can slash these gaps.

How Tall Men Can Fight Back: Strategies to Shorten the Longevity Gap

You can’t shrink yourself or change your gender, but you can mitigate the risks. Drawing from Blue Zones (where 80% of longevity comes from lifestyle, not genes), focus on habits that counter height and gender vulnerabilities. 32 36 In these areas, men match women’s lifespans through community, diet, and activity. 30

1. Boost Heart and Lung Health: Tall frames stress these systems, so aim for 150 minutes of aerobic exercise weekly (walking, swimming) plus strength training 2-3 times a week. 25 This builds efficiency and reduces strain.
2. Anti-Inflammatory Diet: Combat extra cells’ mutation risks with Mediterranean eats—fruits, veggies, nuts, fish, olive oil. Try intermittent fasting for cellular repair. 20 24 Swap junk for produce to add years.
3. Proactive Screenings and Mobility Work: Get annual checks for blood pressure, cholesterol, and cancers (start early as a tall guy). 21 Add yoga or Pilates to fix posture and prevent joint wear—tall people need this extra. 19
4. Social and Stress Management: Build connections (Blue Zone secret) and meditate—men benefit hugely from reducing isolation. 20 Sleep 7-9 hours nightly.
5. Avoid Pitfalls: No smoking, limit alcohol, maintain a healthy weight (your 160 lbs at 6 feet is spot-on). 21 26

These can add 10+ years, potentially closing the gender gap entirely (like in Blue Zones) and halving the height penalty. 24 27

The Residual Gap: What Remains After Your Best Efforts?

Even with all this, biology leaves a mark. The gender gap might vanish in optimized scenarios, but height’s unchangeable aspects (more cells, higher energy needs) could leave tall men 2-4 years behind shorter women or men. 17 22 It’s not inevitable—individual factors rule—but expect a small lingering edge for the short and female.

Wrapping It Up: Take Control of Your Long Game

Longevity isn’t just about height or gender; it’s 80% lifestyle. 32 Tall men, you’ve got hurdles, but with these strategies, you can leap over most of them. Start small: Swap a snack for a walk, book that checkup. Who knows—you might outlive us all. What’s your take? Drop a comment below on your longevity hacks!

Disclaimer: This is general info based on studies; consult a doctor for personalized advice.

Wake up:

Lactoferrin

Creatine 5g

(No because Lactoferrin) Nature’s Way Cell Forté IP-6 & Inositol

(No bc Lactoferrin) Vitamin C 300% DV

(No bc Lactoferrin) CaAKG

Apigenin

Taurine

Glycine 5g + NAC 600mg

Theanine

Hyaluronic Acid

Exercise

Breakfast

Broccoli + black beans + sardine + berries

Glutathione

Evening

Magnesium Citrate

Supercharge Your Mitochondria: A Fun Guide to Boosting Your Cellular Power Plants!

Hey there, energy enthusiasts! Imagine your body as a bustling city, and inside every cell, there’s a powerhouse factory churning out the juice that keeps the lights on. That’s your mitochondria – those tiny, bean-shaped organelles that are basically the unsung heroes of your biology. But here’s the kicker: they’re not just passive batteries. At the heart of their magic is the electron transport chain (ETC), a series of protein complexes (I through IV, plus some sidekicks like II+III and citrate synthase) that turn food into ATP, the ultimate cellular currency. Think of it as a high-stakes relay race where electrons zoom along, pumping out energy while dodging oxidative pitfalls.

If your mito complexes are slacking (like in that hypothetical “everything’s terrible” scenario from a cheek-swab test), you might feel like a phone on 1% battery – foggy, fatigued, and far from fabulous. But fear not! We’re diving into how to optimize each one with science-backed hacks, tasty tips, and a dash of fun. We’ll draw from the latest buzz in 2025 research, like how targeting the ETC could be the next big thing in longevity. 0 Remember, this is educational vibes only – chat with your doc before revamping your routine, especially if supplements are involved.

The Basics: Rev Up Your Mito Engine Before Diving Deep

Before we zoom into each complex, let’s lay the foundation. Mitochondria thrive on lifestyle upgrades that boost biogenesis (making more of ‘em) and efficiency. Here’s your starter pack:

• Move It to Improve It: High-Intensity Interval Training (HIIT) is like a turbo boost for your ETC. Alternate sprints with rests – think 30 seconds all-out, 1 minute chill, for 20 minutes. Studies show it amps up complex activity and even helps T-cells (your immune warriors) by supporting mitochondrial function. 1 Bonus: It fights age-related decline in the chain. 2 18 “LARGE”
• Heat Things Up: Sauna sessions (20-30 minutes at 160-200°F, 2-3 times a week) mimic exercise stress, enhancing ETC complexes and promoting mitophagy (recycling dud mitochondria). It’s like a spa day for your cells – research links it to better heart health and reduced reductive stress. 4 24 “LARGE”
• Shine a Light: Near-infrared red light therapy (10-20 minutes daily via a device) penetrates cells to jazz up complexes, especially by bypassing blocks and boosting ATP. It’s sci-fi cool – think Star Trek healing, but real. Evidence points to it modulating mito function for better energy. 8 12 “LARGE”
• Diet Do’s and Don’ts: Go for a nutrient-dense, Mediterranean-style chow with antioxidants to shield against damage. Avoid inhibitors like excessive manganese or meds (e.g., metformin) that bog down complexes – chat with your doc if you’re on them. 9 Pro tip: Intermittent fasting or a high-carb, low-fat vibe can reset metabolism, but personalize it.

Now, let’s tackle each complex like a boss – with food pics to drool over and tips to try.

Complex I: The Supercharger That Needs a Tune-Up

Picture Complex I as the grand entrance to your ETC party – it handles NADH electrons and can slash ATP by 40% if it’s low. Low vibes? Fatigue city. To max it out:

• Key Nutrients: Riboflavin (B2), iron, and sulfur are your MVPs. B2 acts like a spark plug, iron like the wiring, and sulfur the fuel additive. 3
  • Riboflavin Riches: Load up on dairy, eggs, and almonds. Aim for 1.1-1.3 mg daily from food; supplement 100-400 mg if needed. 3 “LARGE”
  • Iron Boost: Red meat, spinach, lentils – pair with vitamin C for absorption. Supplement wisely (10-40 mg/day) and monitor labs to avoid overload. 6 “LARGE”
  • Sulfur Surge: Eggs, garlic, broccoli – these stink (in a good way) but supercharge. 500-1000 mg from MSM if you’re short. 15 “LARGE”

Fun fact: Avoid caprolactam from sweaty nylon clothes – switch to cotton for mito-friendly workouts!

Complex II: The Succinate Side Door

This one’s your backup electron entry, often suppressed by hypoxia (low oxygen vibes). If it’s sluggish, it can lead to wonky blood vessels or worse. Optimize with:

• Nutrient Squad: Same as I – riboflavin, iron, sulfur. Double down on those foods above.
• Extra Hacks: Cold exposure (ice baths) or keto phases to enhance efficiency. Antioxidants like mitoQ can help, too. 4

Think of it as the understudy – keep it strong so the show goes on!

Complex II+III: The Bridge Builders (Featuring CoQ10)

This duo measures the handoff between II and III, often low due to CoQ10 shortages or blocks. It’s like a traffic jam in your energy highway.

• Star Player: CoQ10: Ubiquinol form, 100-200 mg/day. Foods? Organ meats, fatty fish, spinach. 9 “LARGE”
• Bypass Tricks: If blocked, vitamin C (500-2000 mg), methionine/cysteine (500-1000 mg each from eggs/fish), and that red light therapy. Sulfur amino acids feed straight to IV. 3

Pro tip: Mitochondrial Src might amp Complex I here, but that’s advanced – focus on basics first. 3

Complex IV: The Oxygen Finisher

The grand finale where oxygen turns electrons into water. Low? Poor oxygen use, like running on fumes.

• Cofactor Champs: Copper and iron. Copper from shellfish, nuts, dark chocolate – 1-2 mg/day supplement if low (test first!). 21 “LARGE”
• Boosters: HIIT, selenium (from nuts), and even caffeine/melatonin for subtle tweaks. 8 Models show refining Complex IV math could unlock more, but start simple. 8

Citrate Synthase: The Density Director

Not part of the chain but a TCA cycle marker – high means compensation, low means poor mito density. Optimize with:

• Activity Amps: Exercise (especially endurance) to crank it up. 6 Balanced carbs for acetyl-CoA substrates.
• Holistic Help: NAD+ precursors (NR/NMN, 300-500 mg) and alpha-ketoglutarate (1000-4000 mg) for overall support. 9

Wrapping Up: Your Mito Makeover Awaits

There you have it – a playful playbook to turn your mito complexes from meh to magnificent! Start slow: Nail the basics, track with labs or retests, and watch your energy soar. Who knew cellular science could be this electrifying? If you’re into deep dives, check out emerging therapies like mitochondrial transfers or rapamycin for longevity. 0 2

Disclaimer: This post is for fun and info – not medical advice. Always consult a healthcare pro before changes, especially with supps or if you have conditions. Stay powered up! 🚀

What If We Fixed Almost Everything About Aging… Except DNA Mutations?

Hey folks, imagine this: Scientists wave a magic wand and poof! No more telomere shortening, no rogue senescent cells zombie-ing around, no mitochondrial meltdowns. We cure most causes of aging. How long could we live then?

According to a fascinating new study from researchers at Skolkovo Institute in Moscow (just out on bioRxiv in late November 2025), the answer might be… around 150 years on average. Double today’s lifespan in rich countries! But there’s a catch—these random DNA glitches called somatic mutations would still slowly wreck our cells, especially in the brain and heart. It’s like entropy saying, “Nice try, humans, but I’m still the boss.”

Let’s break this down in a fun, easy way—no heavy math required. Think of it as a video game where aging is the final boss, and mutations are that one unbeatable glitch. 0 “LARGE” 1 “LARGE”

(Cool illustrations of DNA damage piling up over time—those little errors in your cells’ instruction manual that happen just from living.)

Why Mutations Are the Tough Guy of Aging

Your DNA is like the blueprint for your body. Every time cells copy it (or just from wear and tear), tiny mistakes creep in. These are somatic mutations—not the inherited kind, but ones that build up in your body cells over your lifetime.

Some aging problems we might fix someday (like clearing junky cells). But mutations? They’re random chaos—pure disorder (scientists call it “entropy”). Hard to reverse without rewriting your entire genome.

The researchers asked: If mutations were the only aging villain left, how long before they take us down?

They built clever computer models of the human body, treating it like a machine with vital parts: brain, heart, liver, lungs. If one critical part fails… game over. 7 “LARGE” 8 “LARGE”

(Visuals showing how different organs age at different rates—brain and heart are the weak links!)

The “No Aging” Cheat Code: Crazy Long Lives

First, they imagined zero aging at all—just normal risks like accidents. If your death risk stayed frozen at a healthy 30-year-old’s level?

Average lifespan: 430 years. One super-lucky person in billions might hit 14,000 years! (Still not immortal—cars and bad luck exist.) 9 “LARGE” 10 “LARGE”

(Survival curves: Normal aging drops fast after 100. No aging? A gentle slope forever.)

Enter the Mutation Monster

Now add mutations. In brain and heart cells (which don’t divide and replace themselves), errors kill cells slowly. Too many dead neurons or heart cells? Organ failure.

Result: Average lifespan crashes to 169-177 years (brain or heart as the limiter).

But wait—liver and lungs are pros at regenerating! They make new cells to replace damaged ones, so mutations barely slow them down. These organs could last thousands of years.

The big reveal: Your brain and heart are the bottlenecks. They’re like vintage cars with irreplaceable parts.

The Grand Finale: Multi-Organ Showdown

Combining everything, the models predict we’d live 134-170 years on average if mutations were the only problem. That’s huge progress—healthy until 150? Sign me up!

But it also means mutations are a major aging driver (maybe half the battle), and other stuff contributes the rest.

Why This Is Awesome (and a Bit Scary)

This paper is like a roadmap for longevity hunters. It says: Fixing the “reversible” aging stuff could buy us decades more. But to go further, we need breakthroughs against mutations—maybe better DNA repair or protecting key cells.

It’s optimistic yet realistic. We’re not hitting 1,000 years soon, but 150? That could be the prize for the next generation of anti-aging tech.

What do you think—would you want to live to 150 if it meant staying sharp and active? Hit the comments!

Check the full paper here: bioRxiv DOI: 10.1101/2025.11.23.689982. Super readable abstract, and the figures are gold.

Stay young at heart (literally)! 🧬✨

Unlocking the Secrets of Slowing Down Aging: What a New Study Reveals About amino acids and Longevity

Hey there, fellow health enthusiasts! If you’re like me, you’re always on the lookout for science-backed ways to stay youthful, energetic, and healthy as the years tick by. Well, buckle up because a fascinating new study published in Biogerontology (2026) has just dropped some game-changing insights. Titled “Association of Plasma Metabolites with Epigenetic Age Acceleration: A Two-Sample Mendelian Randomization Study,” it dives into how certain chemicals in your blood—called plasma metabolites—might be speeding up (or slowing down) your body’s internal aging clock.

In this blog post, I’ll break it all down in simple terms—no PhD required. We’ll cover what the study found, why it matters, and most importantly, a bunch of actionable steps you can take right now to potentially tweak these metabolites for a longer, healthier life. Think of it as your practical guide to hacking aging through diet, supplements, and lifestyle tweaks. Let’s get into it!

What’s This Study All About? A Quick Primer on Epigenetic Aging

First things first: Aging isn’t just about wrinkles or gray hair. Your “chronological age” is how many birthdays you’ve had, but your “biological age” is how old your cells and body systems really feel. The study focuses on something called epigenetic age acceleration (EAA), which measures how fast your DNA is picking up chemical tags (epigenetic changes) that make you age quicker biologically. Faster EAA is linked to higher risks of diseases like heart disease, cancer, and even a shorter lifespan.

A key highlight? They grouped these metabolites into 13 “metabolic pathways” (think of them as biological assembly lines). The top one was cysteine and methionine metabolism, which emerged as a potential master switch for aging. Other pathways like one-carbon metabolism and tryptophan breakdown also popped up. This isn’t just correlation—the genetic approach suggests these could cause faster aging.

David Sinclair, the renowned longevity expert from Harvard, even shared a figure from the study on X (formerly Twitter) in December 2025, calling out cysteine and methionine as the most significant. The chart shows these pathways ranked by how “enriched” they are in aging-related metabolites, with red bars for the strongest links. Why care? Because tweaking these pathways through everyday habits could help you dial back your biological clock.

The Star Pathway: Cysteine and Methionine Metabolism

This pathway is like a factory handling two key amino acids: methionine (from foods like meat and eggs) and cysteine (which your body makes or gets from diet). It’s crucial for detoxing, making energy, and adding those epigenetic tags to DNA. The study found three metabolites here—methionine, 5-methylthioadenosine, and α-ketobutyrate—tied to faster EAA.

Why it accelerates aging: High methionine levels can lead to excess “methyl groups” messing with DNA, causing inflammation and oxidative stress. In animal studies, cutting methionine extends lifespan by up to 30%! On the flip side, boosting cysteine (via antioxidants like glutathione) might protect against this.

Actionable steps? We’ll cover them below, but the gist is: Restrict methionine without going overboard, and support cysteine for balance.

Other Key Pathways to Watch (and Modulate)

The study didn’t stop at one pathway—13 in total were linked to EAA. While cysteine/methionine is #1, others like one-carbon metabolism (involving folate) and tryptophan metabolism (linked to brain health) are close behind. These often overlap, so addressing one can help others.

• One Carbon Pool by Folate: Handles vitamin B9 for DNA tweaks; excess can imbalance epigenetics.
• Glycine, Serine, and Threonine Metabolism: Supports protein building and detox; low glycine speeds aging.
• Tryptophan Metabolism: Breaks down into inflammatory compounds; high activity worsens neurodegeneration.
• Phenylalanine, Tyrosine, and Tryptophan Biosynthesis: Affects mood chemicals; imbalances shorten lifespan.
• Valine, Leucine, and Isoleucine Biosynthesis: Branched-chain amino acids (BCAAs) that, when too high, promote frailty.
• Lower-ranked ones like sphingolipid metabolism (cell signaling) and purine metabolism (DNA building blocks) suggest avoiding excess fats and monitoring inflammation.

Why modulate these? Dysregulation fuels chronic issues like insulin resistance and cognitive decline. Studies show restricting or balancing them mimics anti-aging diets like fasting.

Your Action Plan: 15+ Steps to Slow Epigenetic Aging

Now for the fun part—practical, do-it-today steps! These are based on the study’s findings, combined with supporting research on longevity. I’ve grouped them by category, with reasons why they work and tips to get started. Remember, these are general suggestions; track your progress with blood tests (e.g., for homocysteine or epigenetic age via kits like TruAge) and chat with a doctor or nutritionist.

Diet Tweaks for Methionine and Amino Acid Restriction

1. Cut Back on High-Methionine Foods: Limit red meat, eggs, dairy, and poultry to 1-2 servings per week. Why? Excess methionine from animal proteins accelerates EAA by disrupting DNA methylation. Action: Swap for plant-based options like beans or tofu—aim for under 1g methionine daily (use an app like Cronometer to track).
2. Go More Plant-Based Overall: Build meals around veggies, fruits, grains, and legumes. Why? Plants are naturally low in methionine and BCAAs, mimicking longevity diets that slow aging in humans and animals. Action: Try a “Meatless Monday” expanded to most days; recipes like lentil soup or veggie stir-fries are easy wins.
3. Restrict BCAAs (Valine, Leucine, Isoleucine): Cap whey protein shakes, eggs, and meat—target 10-15g total BCAAs daily. Why? High BCAAs link to obesity and shorter lifespan; restriction extends life in models. Action: Opt for low-BCAA proteins like rice or peas; avoid BCAA supplements unless you’re an athlete.
4. Limit Tryptophan-Rich Foods: Moderate turkey, bananas, and dairy. Why? Overactive tryptophan metabolism produces inflammatory kynurenine, speeding brain aging. Action: Balance with anti-inflammatory foods like salmon (for omega-3s) once a week.
5. Reduce Phenylalanine and Tyrosine Intake: Cut back on cheese, soy, and almonds. Why? High levels disrupt neurotransmitter balance, contributing to cognitive decline. Action: Choose alternatives like oats or fruits; keep portions small.

Supplements to Support Key Pathways

6. Take N-Acetylcysteine (NAC): Start with 600-1200mg daily, split into doses. Why? Boosts cysteine for antioxidant protection, countering methionine’s aging effects. Action: Buy from a reputable brand; pair with meals to avoid stomach upset.
7. Add Glycine Supplements: 3-5g daily, perhaps in powder form. Why? Balances glycine/serine/threonine pathways, mimicking restriction and aiding methylation. Action: Mix into tea or smoothies; sources like bone broth work too.
8. Incorporate Folate and B Vitamins: 400mcg folate plus B6 (10-50mg), B12 (500mcg), and B2 daily. Why? Optimizes one-carbon metabolism, lowering homocysteine and EAA. Action: Eat folate-rich greens or take a B-complex; get levels tested first.
9. Consider CoQ10 for Ubiquinone Pathway: 100-200mg daily. Why? Supports mitochondrial energy; deficiencies worsen with age, per the study’s lower pathways. Action: Take with fatty meals for better absorption.

Lifestyle Habits to Amplify Benefits

10. Practice Intermittent Fasting: Fast 12-16 hours daily (e.g., eat between 10 AM-6 PM). Why? Synergizes with amino acid restriction, reducing oxidative stress and promoting longevity. Action: Start slow; hydrate during fasts.
11. Monitor Homocysteine Levels: Get a blood test every 6 months. Why? High homocysteine (from methionine) accelerates aging; keeping it under 10 µmol/L protects epigenetics. Action: If high, amp up B vitamins.
12. Exercise Regularly: Mix cardio and strength training 3-5 days a week. Why? Influences amino acid metabolism, balancing pathways like tryptophan and reducing EAA. Action: Walk 30 minutes daily plus weights; it also lowers inflammation.
13. Manage Stress with Meditation: 10-20 minutes daily. Why? Stress disrupts one-carbon pathways; mindfulness slows epigenetic aging. Action: Use apps like Headspace.
14. Avoid Excess Saturated Fats: Limit butter and fried foods. Why? Ties into lipid pathways like sphingolipids, which fuel inflammation when overactive. Action: Choose olive oil or avocados instead.
15. Explore Advanced Options: Try methionine-low meal replacements or consult a pro for personalized plans. Why? For those wanting precision, these target the core pathway without guesswork. Action: Look into apps or books like Sinclair’s Lifespan for more.

Wrapping It Up: Your Path to a Younger You

This study is a reminder that aging isn’t inevitable—we can influence it through smart choices targeting metabolites and pathways. By focusing on methionine restriction, pathway support, and healthy habits, you might not just add years to your life but life to your years. Start with 2-3 steps that fit your routine, track how you feel, and adjust. Remember, results vary, so professional advice is key to avoid deficiencies.

What do you think—ready to give methionine restriction a shot? Drop a comment below if you’ve tried any of these or have questions. Stay youthful, friends!

Disclaimer: This isn’t medical advice. Consult healthcare pros before major changes.

Taking pro-biotic during treatment: Toniiq Probiotic 200 and Supersmart H. Pylori Advanced Formula

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